Hypoglycemic feeling
So, now I'm freaking out that I may be getting Reactive Hypoglycemia. It fits the signs. I also feel like mashed potatoes are a pretty lousy choice for me and maybe my body is just saying "NO" to simple carbs.
What do you think? Any of you get like this? If so, how do I keep it from happening?
Thanks!
I get the giggles when you call me Jessica -- that is actually my surgeon's name. My name is Carlea. I had it show up on my screen name for a while, but it's unique enough that I didn't really want it picked up on with a google search. I like the name Jessica well enough though! :) LOL
I also think that you need to keep track if this happens again after simple carbs.
I havent had any of these episodes yet, but having a hard time just getting the protein down at this point.
Good luck!
Maria
But - I do think I'm getting more sensitive to simple carbs, and it triggers it. So, maybe that's a good thing - makes me think twice before I eat any.
I googled Reactive Hypoglycemia and even found a reference that people who have had RNY are susceptible to developing it...
HW-218/SW-208/CW-126/ Lowest Weight-121/Goal-125 - hit 8/23/09/Height-5'3"
Regain 30 lbs from 2012 to 2016 - got back on track and lost it. Took 8 months.
90+/- pounds lost BMI - 24 or so
Starting BMI between 35 and 40ish?
Join us on the Lightweights Board!
Here is my post from my blog:
After doing research on many aspects of both, the DS is better not just for resolving diabetes but for keeping it away from your door down the road. I spent much time over on the RNY board and time after time, I saw threads about hypoglycemia. So I looked it up on my favorite diabetes format, the endocrinologists that TREAT diabetes.
Here is what I found:
Endocrine News
Patients who undergo Roux-en-Y gastric bypass surgery (RYGBP) experience many benefits such as dramatic weight loss and type 2 diabetes remission. Yet, they also face a risk for developing severe postprandial hypoglycemia due to gastric dumping.Researchers have observed elevated levels of the incretin glucagon-like peptide 1(GLP-1) postsurgery, which has been linked to increased β-cell proliferation and differentiation. A research team led by Josep Vidal, M.D., Ph.D., at the Hospital
Clínic Universitari in Barcelona, Spain, investigated whether a rise in this hormone could over time cause this severe setback.The team divided 24 women into three groups according to time after RYGBP (9–15 mo, 21–30 mo, and > 30 mo). Controls were 8 additional normal weight and 8 morbidly obese women. The subjects fasted overnight and ate a standardized test meal the following day. Blood samples to measure GLP-1, immunoreactive insulin, plasma glucose, and glucagon were taken beforeeating and 10, 30, 60, 90, and 120 minutes after the meal. The patients also underwent an intravenous glucose tolerance test to look for insulin secretion and insulin sensitivity and used a continuous glucose monitoring system to record their postprandial glucose profiles.
In an upcoming article in The Journal of Clinical Endocrinology & Metabolism,* the researchers report that although GLP-1 rose steadily after RYGBP, it did not eventually cause inappropriate insulin secretion. Additionally, their data did not reflect a link between asymptomatic postprandial hypoglycemia in the RYGBP-operated women and an unsuitable relationship between β-cell function and insulin sensitivity. The researchers called for further studies to examine why some patients develop severe postprandial hypoglycemia.
While that article says there wasn’t enough evidence at this time, it did give me pause in that they are even considering the issue.
Then there was a blog article from the EndocrineToday that intrigued me:
Hypoglycemia after Roux-en-Y surgery for weight reduction
Posted by Michael Kleerekoper, MD, MACE April 7, 2009 11:26 AM
Endocrine Today Blog
Seven years ago, my patient had a Roux-en-Y procedure to fight her obesity, and the result was just what she wanted — substantial weight reduction and “no more diabetesÂ" as she reported with a huge smile. Her weight had been stable for a few years, and she was comfortable with it. Four months before her office visit, and for reasons she could not explain, she felt the need to go on a weight-reduction diet during which she lost 12 lb. Two months before she was referred to me, she began to experience episodic hypoglycemia. In her early post-surgery period, she had experienced very typical “dumping syndromeÂ" symptoms, but they had finally cleared and the recent episodes of hypoglycemia seemed quite different.
At 10 p.m. one evening, she felt weak and her capillary blood glucose was 50 mg/dL. This was several hours after dinner. Over the next several weeks, she had a CBG of 53 at 7:45 p.m., 59 at 10:30 p.m., 45 at 3:30 p.m., and most worryingly to her, she woke at 1 a.m. one night feeling very unwell and disoriented, and her CBG was 45 mg/dL. She never experienced fasting hypoglycemia.
Physical examination was essentially normal aside from a suggestion of hyper-pigmentation of her abdominal scar and palmar creases. Pulse and blood pressure were normal as were visual fields and the thyroid examination. Her laboratory findings were also all normal, including electrolytes, fasting blood glucose of (89), insulin, C-peptide, cortisol and adrenocorticotropic hormone.
The history had many characteristics of the dumping syndrome, but several pieces of information did not quite fit. Dumping syndrome is not uncommon in the early months after a Roux-en-Y procedure, but patients generally adapt well by taking frequent very small meals, and over time, the syndrome seems to resolve. Additionally, why did the hypoglycemia occur only several hours after a meal and not sooner? She tried several approaches to changing her eating habits, but these episodes persisted.
I discussed this case with my colleague Dr. Anu Puttagunta, who had cared for a patient with much the same history. This late (post-weight-reduction surgery ) and delayed (post-meal) hypoglycemia has been reported,1, 2 but the mechanism remains elusive as far as I could tell from my reading. The articles reported that some patients did respond to frequent small meals that had little carbohydrate while others only responded when the diet change was accompanied by acarbose.
In some patients, it appears that acarbose alone was sufficient. Dr. Puttagunta’s patient did well with diet modification plus acarbose, so I have begun that same therapy on my patient. She had found those same articles on her own, had modified her diet and had no subsequent episodes of hypoglycemia, but that was not reassuring to her because they were so episodic. When adding the acarbose it was important to remind her to take the tablet (I started with 25 mg three times per day) as soon as she takes her first bite of food. I will report her progress after a few months.
1: Kellogg TA. Surg Obes Relat Dis. 2008;4:492-499. PMID: 18656831.
2: Moreira RO. Obes Surg. 2008;18:1618-1621. PMID: 18566871.
But was was the final straw that pushed me into the DS camp were the guidelines published for the Endocrine Society in March 2009.
Evaluation and Management of Adult Hypoglycemic Disorders:
An Endocrine Society Clinical Practice Guideline
First published in the Journal of Clinical Endocrinology & Metabolism, March 2009, 94(3): 709-728
Hypoglycemia can occur as a result of hyperinsulinism in the absence of previous gastric surgery or after Roux-en-Y gastric bypass for obesity. (pg8)
Some persons who have undergone Roux-en-Y gastric bypass for obesity have endogenous hyperinsulinemic hypoglycemia most often due to pancreatic islet nesidioblastosis, but occasionally due to an insulinoma (48–50). With nesidioblastosis, spells of neuroglycopenia usually occur in the postprandial period and develop many months after bariatric surgery. Spells of neuroglycopenia that occur in the fasting state soon after bariatric surgery are more likely due to a preexisting insulinoma (51). The predominance of women with post-gastric-bypass hypoglycemia may reflect the gender imbalance of bariatric surgery. The precise mechanisms of hypoglycemia remain to be determined (52–54). The incidence of this disorder is unknown, but at the Mayo Clinic the number of cases exceeds, by a considerable degree, that of insulinoma. Partial pancreatectomy is recommended for nesidioblastosis in patients who do not respond to dietary or medical (e.g. an a-glucosidase inhibitor, diazoxide, octreotide) treatments. (pg11)
I checked the document for any mention of the DS and there weren’t any but there were the two references to the RNY.
Bottom line, get a meter, learn to test yourself when you feel like that, tell your surgeon. AND take your meter as proof. Buy and keep glucose tabs with you so if you do go low you can get your blood sugar up as fast as possible.
Also learn to eat about every 2-3 hours, something small and protein driven to keep your insulin on an even keel. Stay away from simple carbs and most other carbs. If your do eat carbs, eat them with protein, such as the carbs in milk, etc.
Liz
Duodenal Switch (Lap) 01-24-11 | Surgeon: Stephen Boyce | High weight: 250 in 2002 | Surgery weight: 203 | Lowest weight: 121 | Current weight: 135 | Goal weight: 135