that would be great i added you as friend.

i am not sure if you saw the post on how IBS actually shows brain differences compared to women that do not have.  the difference was in women who had pain as its primary complaint with IBS.  It is under roux en Y i believe.  if can not find it let me know and i can get it to you.  i even emailed the researchers to see if the same process could be for those after gastric bypass as well.  this just shows the brain gut hypothesis are correct and there is deffinately a change after symptoms so no longer a stress thing and no longer can be said nothing wrong

I also have a high ferritin it's sitting at 465 and has not moved for over a year what does it mean when it's hat high my gp and surgeon do not seem concerned over it

Shrinking Violet , i empathize with you.  i have had almost all of the same problems, testing and surgeries as you.  mym diagnostic lap showed intussusception(where your intestines telescope into themselves) a mesenteric defect and adhesions.  i still have pain and nausea, now throwing up and have a low grade fever now and then(the fever came after my egd with ballon assist).  i feel like my doctors are blowing me off, my surgeon told me i need a psych consult.  i'm not crazy and neither are you or anyone else on this board suffering from abdominal pain, nausea or any complications.  i've been told numerous times by different doctors that "you are high risk" well do something, don't just sit on my symptoms!

please keep us updated, message me, there's alot more i'm sure we can chat about.

ill be sending you a friend request with my personal email so you can email me.

i'll keep you in my prayers

GL with HIDA scan, if for some reason you are really bad off it will not hurt to goto ER.  In mean time start looking for physicians in your area that you can see for second opinion and make sure they are in network for insurance.  By doing some investigation now, and knowing which ones may be viable sources for second opinion will be helpful.  This way you have the info and give to primary care physician with name address phone fax and contact person in office.

Do what you think is right for yourself, listen to you, because only you know really know yourself no one else.

It is difficult when doctors think it all in your head and seem demissive. Letg them know you do not like the demissive tone and that it makes you feel insignificant.  Basically stating you do not like tone used and appreciate that it be used. 

After having gallbladder removed the liver continuously makes bile that is secreted into small intestine and stomach which is extremely irritating causing many digestive issues from nausea, pain, gas, bloating ect.  And yes gallstones can still be formed after the gallbladder is removed.

If the HIDA scan is inconclusive there is always the possibilityy of using a pediatric endoscope and do a modified ERCP and the pediatric endoscope can go further into small intestine and into roux limb so biopsoies can be taken from areas not done before.

Other idea camera endoscopy, even look at issues that may have been preexisting before surgery that manifested worse afterwards.

I will keep you in my thoughts of wellness and hope answers found soon.

It seems not only do I have a lot of the same symtoms of pain. nausea, alot unexplained,  but several here as well.  It varies with each person in the degree which one is affected and impacting of thier life and health.

I came across this the other day and think maybe the same thing can also happen after gastric bypass because of the instantaneous disrtuption in diet and foods eaten shocking the body and the brain whom has a hard time dealing with the sudden change and so goes into survivial mode.

I wonder what has gone on with our bodies after the surgery and possible that the brain chemistry and signals have changed from what happened before to current.  I would love to see a study this point of view and see if there are simularities. 

If simularities exist is the existance or potential there to warrent a closer look before a person has the surgery done, and if none but have what are some precaustions that can be done to not change the brain and its perception afterward?

Study finds structural brain alterations in patients with irritable bowel syndrome

Findings suggest IBS similar to other pain disorders

By Rachel Champeau July 22, 2010
  A large academic study has demonstrated structural changes in specific brain regions in female patients with irritable bowel syndrome (IBS), a condition that causes pain and discomfort in the abdomen, along with diarrhea, constipation or both.    A collaborative effort between UCLA and Canada's McGill University, the study appears in the July issue of the journal Gastroenterology.   The findings show that IBS is associated with both decreases and increases in grey matter density in key areas of the brain involved in attention, emotion regulation, pain inhibition and the processing of visceral information.   IBS affects approximately 15 percent of the U.S. population, primarily women. Currently, the condition is considered by the medical field to be a "functional" syndrome of the digestive tract not working properly rather than an "organic" disorder with structural organ changes. Efforts to identify structural or biochemical alterations in the gut have largely been unsuccessful. Even though the pathophysiology is not completely understood, it is generally agreed that IBS represents an alteration in brain-gut interactions.    These study findings, however, show actual structural changes to the brain, which places IBS in the category of other pain disorders, such as lower back pain, temporomandibular joint disorder, migraines and hip pain — conditions in which some of the same anatomical brain changes have been observed, as well as other changes. A recent, smaller study suggested structural brain changes in IBS, but a larger definitive study hadn't been completed until now.   "Discovering structural changes in the brain, whether they are primary or secondary to the gastrointestinal symptoms, demonstrates an 'organic' component to IBS and supports the concept of a brain-gut disorder," said study author Dr. Emeran Mayer, professor of medicine, physiology and psychiatry at the David Geffen School of Medicine at UCLA. "Also, the finding removes the idea once and for all that IBS symptoms are not real and are 'only psychological.' The findings will give us more insight into better understanding IBS."   Researchers employed imaging techniques to examine and analyze brain anatomical differences between 55 female IBS patients and 48 female control subjects. Patients had moderate IBS severity, with disease duration from one to 34 years (average 11 years). The average age of the participants was 31.   Investigators found both increases and decreases of brain grey matter in specific cortical brain regions.     Even after accounting for additional factors such as anxiety and depression, researchers still discovered differences between IBS patients and control subjects in areas of the brain involved in cognitive and evaluative functions, including the prefrontal and posterior parietal cortices, and in the posterior insula, which represents the primary viscerosensory cortex receiving sensory information from the gastrointestinal tract.   "The grey-matter changes in the posterior insula are particularly interesting since they may play a role in central pain amplification for IBS patients," said study author David A. Seminowicz, Ph.D., of the Alan Edwards Centre for Research on Pain at McGill University. "This particular finding may point to a specific brain difference or abnormality that plays a role in heightening pain signals that reach the brain from the gut."   Decreases in grey matter in IBS patients occurred in several regions involved in attentional brain processes, which decide what the body should pay attention to. The thalamus and midbrain also showed reductions, including a region — the periaqueductal grey — that plays a major role in suppressing pain.   "Reductions of grey matter in these key areas may demonstrate an inability of the brain to effectively inhibit pain responses," Seminowicz said.   The observed decreases in brain grey matter were consistent across IBS patient sub-groups,  such as those experiencing more diarrhea-like symptoms than constipation.   "We noticed that the structural brain changes varied between patients who characterized their symptoms primarily as pain, rather than non-painful discomfort," said Mayer, director of the UCLA Center for Neurobiology of Stress. "In contrast, the length of time a patient has had IBS was not related to these structural brain changes."   Mayer added that the next steps in the research will include exploring whether genes can be identified that are related to these structural brain changes. In addition, there is a need to increase the study sample size to address male-female differences and to determine if these brain changes are a cause or consequence of having IBS.   The study was funded by the National Institutes of Health.   Additional authors include M. Catherine Bushnell, Ph.D., of McGill University, and Jennifer B. Labus, Joshua A. Bueller, Kirsten Tillisch and Bruce D. Naliboff, Ph.D., all of UCLA.   For more news, visit the UCLA Newsroom and follow us on Twitter.

http://newsroom.ucla.edu/portal/ucla/PRN-study-finds-structu ral-brain-alterations-156210.aspx


Here is the orginal source in abstract
http://www.gastrojournal.org/article/S0016-5085(10)00482-8/a bstract