stomache pain after eating/nausea 7yrs pos RNY
I'm hoping someone can help me. I recently just recovered from a gastrointestinal bacteria that hospitalized me for several days. Severe diarrhea/vomiting. I lost about 11 pounds in 4 days; I am now at 99lbs and it appears the bacteria is gone but now I am having severe stomache pain after I eat and nausea to the point of wanting to vomit. I was constipated and now have switched back to diarrhea. I have stuggled for years after my surgery with stomache pain and nausea and always assumed it was just part of having the WLS. I haven't seen my surgeon since 2004. But now as I read some of your posts I wonder....could I have an internal hernia? could this bacteria I had have aggravated it?I called my surgeon today who can't see me until next week but just hoping someone may have some suggestions or what the common symptoms are of an internal hernia????? Any thoughts would be most appreciated.
I'm surprised nobody has posted, and I'm sorry I can't be much help as I haven't had the surgery yet. But from my research and reading the boards, I thnk if you're having nausea that isn't going away, you should play it safe by getting to the doctor. If you're having bloody stool and abdominal pain as well as nausea, then you really should get to the ER, as this is an indication of bowel obstruction. If it's just nasuea, then I think it could be any of the things you listed, or an ulcer. Either way, only the Dr. could give you a diagnosis, so it's good you called the surgeon.
Good luck!
Good luck!
5'6 250/190/140
RNY Scheduled 6/01/10
abbysmommie
on 6/5/10 4:16 am
on 6/5/10 4:16 am
I would love to hear what you find out. I am also 7 years out. I have been essentially constipated since my gastric bypass until this past year when I developed an issue with diarrhea. It has progressed to the point where I have not had a formed BM in months, I have an anal fistula, and now I feel like I have food stuck in my throat but I know I don't because I hardly eat anything and have an acidic feeling. I have been dx with ulcertive colitis. But can't help but think there is more to this story. I have very weird stomach/abd pains and in the past month chest pains that I know are not cardiac.
I am scared that I am breaking down a suture line or something. I do take a lot of medications due to some other medical conditions and I have always viewed it as risk vs benefit...well, I am wondering if it is payment time.
I am scared that I am breaking down a suture line or something. I do take a lot of medications due to some other medical conditions and I have always viewed it as risk vs benefit...well, I am wondering if it is payment time.
I haven't logged on here for months.. and can't believe yours was the first post I read. i recently had surgery for an internal hernia that caused a small bowel obstruction...while 26 weeks pregnant. I am about a year and a half out from lap RNY with no problems what-so-ever until then. It presented as SEVERE stomach pain with nausea/vomiting. It took docs 3 days to figure it out...the baby had everyone confused...it was nothing to do with her! Well, they wised up real quick when I started puking stool...yuck I know. Point is, ever since they fixed the hernia & obstruction I have been having frequent stomach pain and ALOT of nausea. While pregnant, all the docs said was the baby is fine & hopefully it will go away with delivery! I delivered a healthy, beautiful girl 6 weeks ago & I have an appointment w/my surgeon on wed as the symptoms have continued...we shall see....this is miserable!! I was the perfect weight loss surgery patient until the hernia and now I almost wish I had never had it (the weight loss surgery OR the hernia of course!)!!! I am hoping the surgeon can fix me....
It seems not only do I have a lot of the same symtoms but several here as well. I wonder what has gon on with our bodies after the surgery and possible that the brain chemistry and signals have changed from what happened before.
I came across this the other day and think maybe the same thing can also happen after gastric bypass because of the instantaneous disrtuption in diet and foods eaten shocking the body and the brain whom has a hard time dealing with the sudden change and so goes into survivial mode.
A large academic study has demonstrated structural changes in specific brain regions in female patients with irritable bowel syndrome (IBS), a condition that causes pain and discomfort in the abdomen, along with diarrhea, constipation or both. A collaborative effort between UCLA and Canada's McGill University, the study appears in the July issue of the journal Gastroenterology. The findings show that IBS is associated with both decreases and increases in grey matter density in key areas of the brain involved in attention, emotion regulation, pain inhibition and the processing of visceral information. IBS affects approximately 15 percent of the U.S. population, primarily women. Currently, the condition is considered by the medical field to be a "functional" syndrome of the digestive tract not working properly rather than an "organic" disorder with structural organ changes. Efforts to identify structural or biochemical alterations in the gut have largely been unsuccessful. Even though the pathophysiology is not completely understood, it is generally agreed that IBS represents an alteration in brain-gut interactions. These study findings, however, show actual structural changes to the brain, which places IBS in the category of other pain disorders, such as lower back pain, temporomandibular joint disorder, migraines and hip pain — conditions in which some of the same anatomical brain changes have been observed, as well as other changes. A recent, smaller study suggested structural brain changes in IBS, but a larger definitive study hadn't been completed until now. "Discovering structural changes in the brain, whether they are primary or secondary to the gastrointestinal symptoms, demonstrates an 'organic' component to IBS and supports the concept of a brain-gut disorder," said study author Dr. Emeran Mayer, professor of medicine, physiology and psychiatry at the David Geffen School of Medicine at UCLA. "Also, the finding removes the idea once and for all that IBS symptoms are not real and are 'only psychological.' The findings will give us more insight into better understanding IBS." Researchers employed imaging techniques to examine and analyze brain anatomical differences between 55 female IBS patients and 48 female control subjects. Patients had moderate IBS severity, with disease duration from one to 34 years (average 11 years). The average age of the participants was 31. Investigators found both increases and decreases of brain grey matter in specific cortical brain regions. Even after accounting for additional factors such as anxiety and depression, researchers still discovered differences between IBS patients and control subjects in areas of the brain involved in cognitive and evaluative functions, including the prefrontal and posterior parietal cortices, and in the posterior insula, which represents the primary viscerosensory cortex receiving sensory information from the gastrointestinal tract. "The grey-matter changes in the posterior insula are particularly interesting since they may play a role in central pain amplification for IBS patients," said study author David A. Seminowicz, Ph.D., of the Alan Edwards Centre for Research on Pain at McGill University. "This particular finding may point to a specific brain difference or abnormality that plays a role in heightening pain signals that reach the brain from the gut." Decreases in grey matter in IBS patients occurred in several regions involved in attentional brain processes, which decide what the body should pay attention to. The thalamus and midbrain also showed reductions, including a region — the periaqueductal grey — that plays a major role in suppressing pain. "Reductions of grey matter in these key areas may demonstrate an inability of the brain to effectively inhibit pain responses," Seminowicz said. The observed decreases in brain grey matter were consistent across IBS patient sub-groups, such as those experiencing more diarrhea-like symptoms than constipation. "We noticed that the structural brain changes varied between patients who characterized their symptoms primarily as pain, rather than non-painful discomfort," said Mayer, director of the UCLA Center for Neurobiology of Stress. "In contrast, the length of time a patient has had IBS was not related to these structural brain changes." Mayer added that the next steps in the research will include exploring whether genes can be identified that are related to these structural brain changes. In addition, there is a need to increase the study sample size to address male-female differences and to determine if these brain changes are a cause or consequence of having IBS. The study was funded by the National Institutes of Health. Additional authors include M. Catherine Bushnell, Ph.D., of McGill University, and Jennifer B. Labus, Joshua A. Bueller, Kirsten Tillisch and Bruce D. Naliboff, Ph.D., all of UCLA. For more news, visit the UCLA Newsroom and follow us on Twitter.
http://newsroom.ucla.edu/portal/ucla/PRN-study-finds-structu ral-brain-alterations-156210.aspx
I came across this the other day and think maybe the same thing can also happen after gastric bypass because of the instantaneous disrtuption in diet and foods eaten shocking the body and the brain whom has a hard time dealing with the sudden change and so goes into survivial mode.
Study finds structural brain alterations in patients with irritable bowel syndrome
Findings suggest IBS similar to other pain disorders
By Rachel Champeau July 22, 2010A large academic study has demonstrated structural changes in specific brain regions in female patients with irritable bowel syndrome (IBS), a condition that causes pain and discomfort in the abdomen, along with diarrhea, constipation or both. A collaborative effort between UCLA and Canada's McGill University, the study appears in the July issue of the journal Gastroenterology. The findings show that IBS is associated with both decreases and increases in grey matter density in key areas of the brain involved in attention, emotion regulation, pain inhibition and the processing of visceral information. IBS affects approximately 15 percent of the U.S. population, primarily women. Currently, the condition is considered by the medical field to be a "functional" syndrome of the digestive tract not working properly rather than an "organic" disorder with structural organ changes. Efforts to identify structural or biochemical alterations in the gut have largely been unsuccessful. Even though the pathophysiology is not completely understood, it is generally agreed that IBS represents an alteration in brain-gut interactions. These study findings, however, show actual structural changes to the brain, which places IBS in the category of other pain disorders, such as lower back pain, temporomandibular joint disorder, migraines and hip pain — conditions in which some of the same anatomical brain changes have been observed, as well as other changes. A recent, smaller study suggested structural brain changes in IBS, but a larger definitive study hadn't been completed until now. "Discovering structural changes in the brain, whether they are primary or secondary to the gastrointestinal symptoms, demonstrates an 'organic' component to IBS and supports the concept of a brain-gut disorder," said study author Dr. Emeran Mayer, professor of medicine, physiology and psychiatry at the David Geffen School of Medicine at UCLA. "Also, the finding removes the idea once and for all that IBS symptoms are not real and are 'only psychological.' The findings will give us more insight into better understanding IBS." Researchers employed imaging techniques to examine and analyze brain anatomical differences between 55 female IBS patients and 48 female control subjects. Patients had moderate IBS severity, with disease duration from one to 34 years (average 11 years). The average age of the participants was 31. Investigators found both increases and decreases of brain grey matter in specific cortical brain regions. Even after accounting for additional factors such as anxiety and depression, researchers still discovered differences between IBS patients and control subjects in areas of the brain involved in cognitive and evaluative functions, including the prefrontal and posterior parietal cortices, and in the posterior insula, which represents the primary viscerosensory cortex receiving sensory information from the gastrointestinal tract. "The grey-matter changes in the posterior insula are particularly interesting since they may play a role in central pain amplification for IBS patients," said study author David A. Seminowicz, Ph.D., of the Alan Edwards Centre for Research on Pain at McGill University. "This particular finding may point to a specific brain difference or abnormality that plays a role in heightening pain signals that reach the brain from the gut." Decreases in grey matter in IBS patients occurred in several regions involved in attentional brain processes, which decide what the body should pay attention to. The thalamus and midbrain also showed reductions, including a region — the periaqueductal grey — that plays a major role in suppressing pain. "Reductions of grey matter in these key areas may demonstrate an inability of the brain to effectively inhibit pain responses," Seminowicz said. The observed decreases in brain grey matter were consistent across IBS patient sub-groups, such as those experiencing more diarrhea-like symptoms than constipation. "We noticed that the structural brain changes varied between patients who characterized their symptoms primarily as pain, rather than non-painful discomfort," said Mayer, director of the UCLA Center for Neurobiology of Stress. "In contrast, the length of time a patient has had IBS was not related to these structural brain changes." Mayer added that the next steps in the research will include exploring whether genes can be identified that are related to these structural brain changes. In addition, there is a need to increase the study sample size to address male-female differences and to determine if these brain changes are a cause or consequence of having IBS. The study was funded by the National Institutes of Health. Additional authors include M. Catherine Bushnell, Ph.D., of McGill University, and Jennifer B. Labus, Joshua A. Bueller, Kirsten Tillisch and Bruce D. Naliboff, Ph.D., all of UCLA. For more news, visit the UCLA Newsroom and follow us on Twitter.
http://newsroom.ucla.edu/portal/ucla/PRN-study-finds-structu ral-brain-alterations-156210.aspx
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